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Wouldn't the antibodies attack Humalin R insulin then
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I think this is where my original thought processes were going.
Some people have a gene that causes antibodies against the pancreatic cells, some in addition may have antibodies to insulin. These latter are most likely to develop diabetes early in life (classic juvenile type 1)These not only have to replace the insulin but their body reacts in some way against the insulin. Others,(who tend to develop diabetes later) replace the naturally produced insulin with injected insulin which then works as it should.
Reading on this site I seem to get the impression that my own diabetes and that of many others with LADA is not subject to the erratic swings of BS that many type 1s seem to experience. Its normally explained by reference to residual insulin production which might or might not disappear.
If this genetic data is correct then there are real differences, more than one type of autoimmune diabetes. I haven't found anything to suggest this elsewhere
