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The Flow Theory of Cholesterol and Heart Disease

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xMenace

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I don't know if this concept has a name, but I need to call it something. We've all heard about LDL being the bad cholesterol and HDL being the good cholesterol. These lipoproteins are not cholesterol at all but merely carriers. LDL takes cholesterol to cells from the liver and HDL returns cholesterol from cells to the liver. It's a flowing system.

 

Why does it need a blog entry? Because we haven't got it right. We say simple things like "too much LDL is bad." The problem is, that doesn't cover all the bases, sorry non baseball fans.

 

What got me thinking about this angle are the fruitarians. Personally I can't stand their cult-like behavior, at least the ones I've read. I'm sure there's a few good apples in the bunch. I have a hard time believing their "interpretation" of science. I see it as "it works for me, so it must be true." We all know that science has to cover all the bases.

 

But what if the Fruitarians and Atkinsers are both right about heart disease? Can they be? Can we explain it? Obviously if we are both right, it has to be explainable. We just have to figure it out. I think this flow theory might just do that.

 

So what is this flow theory? I can't give you all the gory details, but guys like Chris Masterjohn can: High Cholesterol and Heart Disease — Myth or Truth? I think what he's saying is that the load of evidence points to oxidyzing LDL as the number one suspect. But things like too much saturated fat, high LDL alone, and physcal experiments do not support the concept that LDL by themselves are in any way dangerous. These are not simple proteins with cholesterol particles stuck to it but complex compounds. One of the things they contain are antioxidants, things that prevent oxydyzation. Further complicating matters are factors such as particle sizes and fat composition -- he says PUFAs are much more prone to oxydization that SFs. His premise is basically this: the longer an LDL is out there floating around, the more it decays. I think we can also add a few others: the less antioxidants packaged in the LDL, the faster it will decay, the more prone the fats contained in it are to oxydization, the faster it will decay, and the smalle they are, the more likely they will get stuck and decay. So lets take a look at these things under three different diets; let's try to measure each diet's atherominity according to this idea of flow or time: the Standard American Diet (SAD), the Atkins (HFLC), and the Fruitarian (F). We'll simply mark them as 0 for good, 1 for indifferent, and 2 for bad. The higher the score, they worse they are.

 

- - - - - - - - 1 HDL qty - - - - - 2 LDL qty - - - - 3 LDL Size - - - 4 PUFA - - - 5 Anti - - - - - - Total

SAD - - - - - - 2 - - - - - - - - - - - 2 - - - - - - - - - 2 - - - - - - - - - 2 - - - - - - - 2 - - - - - - - - 10

HFLC - - - - - - 0 - - - - - - - - - - - 2 - - - - - - - - - 0 - - - - - - - - - 0 - - - - - - - 1 - - - - - - - - - 3

F - - - - - - - - 2 - - - - - - - - - - - 0 - - - - - - - - - 2 - - - - - - - - - 1 - - - - - - - 0 - - - - - - - - - 5

 

Assumptions:

1. Fructose and excess carb consumption cause high triglycerides which borrow APO B-100s from HDLs thus lowering HDL. Less HDL means LDL stay longer.

2. HFLC will add dietary cholesterol. More LDL means more to clear out so they stay longer.

3. Sugar consumption lowers LDL size. The smaller things are, the stickier they get. The more they stick, the longer they stay stuck.

4. HFLCers eat very little PUFAs while F's avoid SFs but also lower fat intake (affects #2 as well).

5. HFLC lowers need for anti-oxidants (because they work better?) and get many thru high veggie consumption; while F consume more. Add 1 to HFLC if u disagree.

* My weightings are even because I don't kow how to weight them sensically. I debate keeping the size factor.

* This analysis addresses only heart disease forces. I still believe obese people suffer from too much carb consumption for their level of insulin resistance.

 

Conclusion

 

It seems according to this flow theory, both low carbers and fruitarians might be right about diet and heart disease. The sure way to fail seems to be SAD. But I'm not a scientist. Don't believe anything I write about this stuff. ;)

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